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To evaluate the total IL-6 level in the wound site, immunofluorescence labeling was performed

Moreover, ACTL6A overexpression relieved the inhibited GC cell proliferation caused by ACTL6A expression knockdown (Supplementary Fig 1c, d)

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However, rivaroxaban has been shown to promote antitumor immunity by enhancing the infiltration of dendritic cells and cytotoxic T cells at the tumor site, blocking FXa PAR-2 signaling [125]